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190-522 - Deploying Domino R5 Applications - Dump Information

Vendor	:	Lotus
Exam Code	:	190-522
Exam Name	:	Deploying Domino R5 Applications
Questions and Answers	:	46 Q & A
Updated On	:	December 6, 2017
PDF Download Mirror	:	190-522 Brain Dump
Get Full Version	:	Pass4sure 190-522 Full Version

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190-522 Questions and Answers

QUESTION 42:

Which one of the following might Günter, a Notes user lose in the process of recreating his NOTES.INI file?

Tasks
User ID
Profile documents
ENVIRONMENT variables Answer: D

QUESTION 43:

Users have complained to Jeff that the views in the Sales.nsf database are taking a long time to open.

Which one of the following Notes.ini parameters can he modify to increase the view buffer size and improve performance?

Buffersize=
BufferPoolSize=
NSFBUFFERPOOLSIZE=
NSF_BUFFER_POOL_SIZE=

Answer: D

"NSF_BUFFER_POOL_SIZE= " in Notes.ini setting will improve the performance when the View takes long time to display.

Search the Domino 5 admin help with "NSF_BUFFER_POOL_SIZE".

QUESTION 44:

A database has been handed off for deployment on Server

A. If Freya has Author access

to that database, which one of the following must be included to allow her to modify the documents that she created?

Nothing
Authors field
Readers field
Editors field Answer: B QUESTION 45:

Danny, the system administrator, wants to allow browse1r4 clients accessing a

Domino-hosted Web site to run a Java applet that uses COBRA. Which one of the following must Bertha do to accomplish this?

Set the database property "Web access: Enable Java Applets".
Start the Domino ORB and enable the IIOP port in the Server document.
Enable the SSL port in the Server document so that the IIOP port can communicate securely with the Java applet.
Nothing. Domino 5.0 natively enabled the execution of Java applets as long as they were complied with the lotus.notes.noi package.

Answer: B

QUESTION 46:

There is a database with many attachments.

Which one of the following replication settings will limit the attachments that are replicated to the sales people's laptops?

Receive summary and 40 KB of rich text only on the server replica.
Receive summary and 40 KB of rock text only on each of the workstation's replicas.
Replicate a subset of documents and choose the view that does not display the attachments in the server replica.
Replicate a subset of documents and choose the view that does not display the attachments in the workstation replica.

Answer: B

Lotus 190-522 Exam (Deploying Domino R5 Applications) Detailed Information

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Lotus 190-522

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getting read 190-522y for Deploying Domino R5 applications look at variousi'm preparing for verify 190*522, "Deploying Domino R5 purposes". i'm a fundamental R5 CLP App Dev, fundamental R4 CLP App Dev, CLP R4 Sys Admin, and have handed my first two R5 Sys Admin exams. i have been a Notes developer and Administrator seeing that 1993. What areas should I brush up in to put together for this test, and what (free) elements are there in the means of look at checks?
First, download the examination book from http://www.lotus.com/certification - this lists the areas of competency for this examination, (i'd encompass those right here, nevertheless it is rather an intensive listing.)
in fact, this examination measures your capabilities of the operation, deployment, administration and maintenance as it relates to databases, rather than to selected server operations similar to routing mail, etc.
you should definitely have a sound expertise of database design facets (given your existing qual's this may still be good enough) and how these design points affect administration alternate options, ie, can the server run brokers in line with current signatures within the app (security!! - hint). remember to even have a sound knowledge of the COMPACT, DESIGN, FIXUP, replace and UPDALL initiatives - what they do, how they function, once they run, etc. Replication additionally points during this examination, although it's center of attention is more from a DB perspective, eg, Mary has a DB on Server A and a rep on Server B, how should still the ACL be modified so that alterations are most effective replicated from Server B to Server A - that form of issue. i might advocate that you turn into fully conversant with all the alternatives, switches and "tell" commands available for these initiatives, eg what would a "Load Compact -r" do? What determine box would you opt for in the projects alternative box when activated from DomAdmin, and so on.
protection and user interaction are also key points during this exam, so you should definitely be entirely conversant with ACL's, AL's (kind/view), read 190-522er/author fields and encryption of fields.
i'm not aware about any free substances for this examination. The LAEC course "Deploying Domino purposes" is a finished e book to the material coated inside this exam and that i would suggest that direction to you. furthermore, self test utility have eventually released a convention check for this exam, which may also be discovered athttp://www.selftestsoftware.com/Product.asp?dept%5Fid=302∏uctpercent5Fid=522 .
One final item, the "Managing Domino Databases" yellow-e-book protected within the Admin Doc-pack is an excellent aid for this exam, examine to peer if any one for your company alread 190-522y has the pack and in that case, examine this book from cowl-to-cover.
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The facts base assisting the management of acute respiratory misery syndrome (ARDS) multiplied considerably in 2014. β-Agonists held out advantage for ARDS by reducing fluid transport across the alveolar membrane through activation of basolateral Na+,k+-ATPase. although, perioperative β-agonists did not reduce the possibility of setting up ARDS in high-chance surgical patients (1). youngsters this therapeutic strategy has did not improve result—perhaps through unanticipated consequences on nonpulmonary organ methods in the presence of severe systemic irritation—choice processes to fighting pulmonary edema can also nevertheless be of improvement. The lectin-like area of tumor necrosis aspect turned into shown to at once spark off the epithelial sodium channel located on the epithelial surface of type II alveolar cells, thereby mitigating the development of pulmonary edema in keeping with lung injury (2). This novel mechanism of alveolar liquid clearance may open up new opportunities for therapeutic intervention, however the success of such interventions depends upon the as yet unclear causal function played by means of pulmonary edema per se in the scientific effect of ARDS.
Statins have been well-known to exert antiinflammatory effects in animal models of sepsis and lung damage (three), and many observational reports discovered that statin remedy become associated with a reduced chance of mortality in sufferers with sepsis (4). In 2014, two enormous randomized trials testing the efficacy of statins for patients with ARDS had been published: in one trial, patients with early ARDS were randomized to simvastatin or placebo (5), whereas within the different trial, patients with sepsis-associated ARDS were randomized to rosuvastatin or placebo (6). both trials discovered no facts of mortality discount from statin remedy; indeed, rosuvastatin was linked to an expanded possibility of renal and hepatic organ dysfunction. These findings suggest that, regardless of their putative antiinflammatory results, statins don't tremendously modulate the mechanisms leading from pulmonary inflammation to demise from ARDS.
Statins and β-agonists be a part of a protracted listing of failed pharmacological therapies for ARDS. These negative consequences, bought despite a sound primary intent for therapeutic effectiveness (7), imply that the causal pathway leading to loss of life from ARDS requires additional careful delineation. Discrepancies between the consequences bought from early fundamental and scientific stories versus the findings of properly powered scientific trials of those healing procedures fortify the suggestion that surrogate result measures in response to organ characteristic can not reliably predict clinical advantage. This truth is above all primary to the examine of ARDS prevention, considering that ARDS itself constitutes a sort of organ characteristic surrogate result (eight). ARDS prevention suggestions should be evaluated in terms of their have an effect on on affected person-headquartered medical consequences in place of their effect on the building of the physiological and scientific elements that constitute ARDS (9).
Keratinocyte growth ingredient is a strong, fibroblast-produced stimulant of epithelial phone increase that exerts a number theoretically advisable consequences in the injured lung (10, eleven), and holds promise as a pharmacological remedy for ARDS. In people challenged with inhaled lipopolysaccharide after pretreatment with palifermin (modified human keratinocyte growth component) versus control, bronchoalveolar lavage fluid confirmed improved type II alveolar telephone proliferation and accelerated tiers of antiinflammatory and proreparative cytokines—a sample in step with enhanced epithelial fix. Palifermin changed into neatly tolerated. Questions in regards to the beneficial dose and route of birth and effectiveness of the agent in sufferers with clinically significant lung damage and pulmonary inflammation stay (10), however additional analyze is naturally warranted.
The incidence of a few standard acute and chronic lung illnesses—together with ARDS, continual obstructive pulmonary sickness, and idiopathic pulmonary fibrosis—rises greatly with age, exceptionally above forty years of age (12, 13). This may arise partially as a result of age-linked changes in the feature of bone marrow–derived mesenchymal stem cells (MSCs). Epithelial restore after lung injury is significantly elegant on MSCs, which mobilize a amazing antiinflammatory response to pulmonary injury. Observations suggest that aged MSCs reveal reduce expression tiers of genes for a couple of key cytokine and chemokine receptors, impairing MSC activation and migration in response to pulmonary inflammation (14). Age-connected changes in tissue elastic houses and within the immune response to damage can also boost the risk of lung harm in older patients (15). These considerations imply that limiting tidal quantity and plateau pressure to evade ventilator-precipitated lung harm (VILI) is specifically critical in older sufferers. then again, besides the fact that children there aren't any randomized trials of lung-shielding air flow options in toddlers, observational reviews in pediatric sufferers suggest a improvement from the use of lessen tidal volumes (15). Given the existing evidence, lung-protecting air flow thoughts are hence likely a good idea in all mechanically ventilated sufferers, in spite of age.
VILI may result from intraoperative mechanical ventilation and can have an impact on perioperative effects (16). however, the PROVHILO (defensive ventilation the use of high versus Low PEEP) trial found no change within the expense of postoperative pulmonary problems with bigger (12 cm H2O) versus decrease fine end-expiratory force (PEEP) (≤2 cm H2O) titration strategies applied all over ordinary anesthesia (17). This finding provides to the checklist of smartly-designed randomized trials failing to exhibit improvement from air flow with higher ranges of PEEP. One cause of the apparent lack of benefit—within the face of a powerful biological cause for advantage—is that patients fluctuate considerably in how they respond to PEEP. as an instance, the growth in oxygenation after a rise in PEEP varies greatly throughout sufferers with ARDS (18); sufferers with significant improvement in oxygenation after an increase in PEEP had reduce mortality than sufferers devoid of improvement in a secondary evaluation of two randomized trials of higher versus reduce PEEP (18). however enhanced oxygenation per se is not going to explain the change in mortality, the oxygenation response might also replicate the diploma of lung recruitment and favorable adjustments in stress and stress on the alveolar level. In some sufferers, PEEP recruits collapsed alveoli and improves the homogeneity of ventilation (19). local air flow inhomogeneities can amplify alveolar stress, propagating VILI (20). The magnitude of such inhomogeneity become linked to sickness severity and outcome in ARDS (19). in addition, lung recruitability was linked to more advantageous discount rates in inhomogeneity. These insights support the critical significance of evaluating the affect of PEEP in individual patients on the bedside to determine its impact on VILI.
Extracorporeal membrane oxygenation (ECMO) is transforming into swiftly in its international application and availability for sufferers with severe acute respiratory failure. Given the complicated infrastructure and scientific services required to securely and with no trouble provide ECMO, there is increasing challenge to be sure that ECMO care is centralized in tertiary facilities with satisfactory event and components. In 2014, a world consortium of leaders in ECMO (ECMONet) launched a consensus commentary recommending that ECMO be offered in high-extent regional or countrywide facilities of excellence (21). The observation particular that ECMO courses should have ample entry to advanced clinical assist capabilities, adequate staffing, and based plans for certifying and holding personnel practicing and conducting pleasant assurance. expanding the homogeneity of care brought to patients with severe acute respiratory failure will additionally facilitate vital analysis into the influence of ECMO on medical and financial outcomes.
identifying whether to provoke ECMO for sufferers with extreme ARDS continues to be a difficult determination balancing the chance of attaining an outstanding influence in a severely sick patient in opposition t the fees linked to using a scarce and aid-intensive intervention. A prognostic instrument, the RESP score, could support in scientific decision-making. The RESP (Respiratory ECMO Survival Prediction) ranking become developed from records in the Extracorporeal existence help corporation registry and externally validated to foretell the chance of survival after initiation of ECMO for extreme ARDS (22) with brilliant calibration and inexpensive discrimination (purchasable at www.respscore.com). The exact medical usefulness of the rating is still uncertain—the score predicts the chance of survival in sufferers in whom ECMO is initiated but doesn't predict even if ECMO initiation will drastically alter the probability of survival (23). furthermore, the decision to provoke ECMO accommodates many extra components together with the trajectory of illness and the affected person’s wishes and values. Clinicians will proceed to rely closely on clinical journey and private judgment in making these complex and time-compelled decisions.
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(2) the usage of the steam tables energy balance: 10 ninety five 10

Unformatted textual content preview: (2) using the steam tables energy steadiness: 10 95 10 397 88 10 = ( 29 = =-( 29 ( 29-( 29 $ . $ $ U T x U T xU T L L V both at saturation C 1 2 forty four three 44 additionally P P T = ( 29 sat and V x V T xV T = =-( 29 ( 29+ ( 29 0 001 10 . $ $ m three L V method i) wager T , get P T sat ( 29 , $ V T L ( 29 , $ V T V ( 29 , $ U T L ( 29 and $ U T V ( 29 from steam tables ii) See if Eqns. (1) and (2) are convinced by using Eqn. (2) to get x , and then seeing if Eqn. (1) is convinced. for example, wager T = eighty C : $ . V L 3 m g = -1029 10 6 $ . U L J g = 334 86 $ . V V three m g = -3407 10 three $ . U V J g = 2482 2 P = 4739 . kPa ( 29 ( 29 = x eqn. 2 029058 . ; x eqn. 1 ( 29 ( 29 = 0 29348 . solutions to Chemical and Engineering Thermodynamics, 3e via iteration and interpolation T = 794 . C , P = 04739 . bar and x = 029 . grams . change between this answer and the old one is because of the inaccuracies of the approximate vapor drive equation partially 1, and the belief of steady warmth capacities. 5.39 the entire P-V statistics for this issue turned into obtained with a simple basic language software written for this difficulty. Calculations were achieved for n-butane as a representative fluid. The van der Waals loop vicinity is proven on the diagram. what is enjoyable is that, in addition to the van der Waals loop, there is tons constitution within the P-V plot. lots of it occurs within the location of b V and V < 0, so that it has no relevance to our calculations. in the region V b there is just the van der Waals loop behavior at low reduced temperatures, and the hyperbolic conduct PV RT = ( 29 at very excessive temperatures. The main point is that the cubic equations we use demonstrate reasonably complex P-V conduct, however handiest distinctly primary conduct in the vicinity of interest to us, which is V b . P-V diagram for n-butane calculated with the Peng-Robinson equation of state for realizable ( V b ) and physically unrealizable ( V b < ) regions. options to Chemical and Engineering Thermodynamics, 3e 5.forty Let T E = the equilibrium transition temperature when each strong phases are good. dG VdP SdT =-additionally dH TdS VdP = + so at steady pressure S T T H T C T P P p F H G I k J = F H G I okay J = 1 phase with better warmth means could have a higher entropy considering that the entropy of each phases are zero at 0 okay. Then, again at consistent power G T S P F H G I k J = -on account that both phases have the equal Gibbs free power at the temperature, T , this means that the substance with the higher entropy (which arises from greater warmth ability) may have the decrease Gibbs free power, and therefore be the strong section. 5.forty one PV RT B T V = + ( 29 1 ; P RT V B T RT V = + ( 29 2 P V RT V T F H G I okay J = -2 ; -( 29 < 2 3 B T RT V 2 1 3 2 B T V V ( 29-; B T V V V ( 29-= -3 2 2 2 ; B T V ( 29-2 lower back to virial eq....View Full doc

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